POLYMORPHISM OF THE SYNDROME OF HYPERACTIVE URINARY BLADDER IN PATIENTS WITH ACUTE AND CHRONIC VASCULAR DISEASES OF THE BRAIN

Abstract

Abstract. The paper discusses the main etiological, phenomenological and pathogenetic mechanisms of forming the syndrome of overactive bladder (OAB) in patients with acute and chronic cerebrovascular diseases. Describes the role of melatonin, arginine vasopressin (AVP) and corticotropin-releasing factor hormone (CRFH) in maintaining the rhythms of urination, urine formation and retention of urine in norm and abnormalities in these systems in acute and chronic vascular pathology of the brain. Described phenomenology OAB syndrome in vascular diseases of the brain. Shows a differentiated approach to pharmacological correction in patients with different clinical variants of urinary disorders and urine formation in the framework of the OAB syndrome with the use of neurotransmitter therapy and hormonesensitive.

Keywords

• cerebrovascular disease
• ischemic stroke
• vascular dementia
• overactive bladder
• melatonin
• arginine-vasopressin
• desmopressin
• corticotropin-releasing factor hormone