mSystems
(Aug 2021)
Recurrent Vulvovaginal Candidiasis: a Dynamic Interkingdom Biofilm Disease of
<i>Candida</i>
and
<i>Lactobacillus</i>
Emily McKloud,
Christopher Delaney,
Leighann Sherry,
Ryan Kean,
Shanice Williams,
Rebecca Metcalfe,
Rachael Thomas,
Riina Richardson,
Konstantinos Gerasimidis,
Christopher J. Nile,
Craig Williams,
Gordon Ramage
Affiliations
Emily McKloud
School of Medicine, Dentistry and Nursing, College of Medical, Veterinary and Life Sciences, Glasgow, UK
Christopher Delaney
School of Medicine, Dentistry and Nursing, College of Medical, Veterinary and Life Sciences, Glasgow, UK
Leighann Sherry
School of Life Sciences, Glasgow, UK
Ryan Kean
Department of Biological and Biomedical Sciences, School of Health and Life Sciences, Glasgow Caledonian University, Glasgow, UK
Shanice Williams
School of Medicine, Dentistry and Nursing, College of Medical, Veterinary and Life Sciences, Glasgow, UK
Rebecca Metcalfe
Sandyford Sexual Health Service, NHS Greater Glasgow and Clyde, Glasgow, UK
Rachael Thomas
Sandyford Sexual Health Service, NHS Greater Glasgow and Clyde, Glasgow, UK
Riina Richardson
ORCiD Division of Infection, Immunity and Respiratory Medicine, Faculty of Biology, Medicine and Health, University of Manchester, Manchester, UK
Konstantinos Gerasimidis
School of Medicine, Dentistry and Nursing, College of Medical, Veterinary and Life Sciences, Glasgow, UK
Christopher J. Nile
School of Dental Sciences, University of Newcastle, Newcastle upon Tyne, UK
Craig Williams
School of Medicine, Dentistry and Nursing, College of Medical, Veterinary and Life Sciences, Glasgow, UK
Gordon Ramage
ORCiD School of Medicine, Dentistry and Nursing, College of Medical, Veterinary and Life Sciences, Glasgow, UK
DOI
https://doi.org/10.1128/mSystems.00622-21
Journal volume & issue
Vol. 6,
no. 4
Abstract
Read online
RVVC is a significant burden, both economically and for women's health, but its prevalence is poorly documented globally due to the levels of self-treatment. Identifying triggers for development and recurrence of VVC and the pathogenesis of the microbes involved could considerably improve prevention and treatment options for women with recurrent, azole-resistant cases.
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