Department of Oral and Molecular Microbiology, Osaka University Graduate School of Dentistry, Osaka, Japan
Satoshi Nagase
Department of Clinical Laboratory Science, Faculty of Health Sciences, Institute of Medical Pharmaceutical and Health Sciences, Kanazawa University, Kanazawa, Japan
Yuki Takahara
Department of Oral and Molecular Microbiology, Osaka University Graduate School of Dentistry, Osaka, Japan
Mariko Honda-Ogawa
Department of Oral and Molecular Microbiology, Osaka University Graduate School of Dentistry, Osaka, Japan
Yasushi Mori
Department of Oral and Molecular Microbiology, Osaka University Graduate School of Dentistry, Osaka, Japan
Yukako Akamatsu
Department of Oral and Molecular Microbiology, Osaka University Graduate School of Dentistry, Osaka, Japan
Department of Clinical Laboratory Science, Faculty of Health Sciences, Institute of Medical Pharmaceutical and Health Sciences, Kanazawa University, Kanazawa, Japan
Shigetada Kawabata
Department of Oral and Molecular Microbiology, Osaka University Graduate School of Dentistry, Osaka, Japan
Secondary bacterial pneumonia following an influenza A virus (IAV) infection is a major cause of morbidity and mortality. Although it is generally accepted that preceding IAV infection leads to increased susceptibility to secondary bacterial infection, details regarding the pathogenic mechanism during the early stage of superinfection remain elusive.
