Data on inheritance of race 2 anthracnose resistance in watermelon (Citrullus spp.) biparental mapping populations
Bed Prakash Bhatta,
Edgar Correa,
Takshay Patel,
Todd C. Wehner,
Kevin M. Crosby,
Michael J. Thomson,
Subas Malla
Affiliations
Bed Prakash Bhatta
Department of Horticultural Sciences, Texas A&M University, College Station, TX 77843, USA; Texas A&M AgriLife Research and Extension Center, 1619 Garner Field Road, Uvalde, TX, 78801, USA
Edgar Correa
Department of Horticultural Sciences, Texas A&M University, College Station, TX 77843, USA; Texas A&M AgriLife Research and Extension Center, 1619 Garner Field Road, Uvalde, TX, 78801, USA
Takshay Patel
Department of Horticultural Science, North Carolina State University, Raleigh, NC 27695, USA
Todd C. Wehner
Department of Horticultural Science, North Carolina State University, Raleigh, NC 27695, USA
Kevin M. Crosby
Department of Horticultural Sciences, Texas A&M University, College Station, TX 77843, USA
Michael J. Thomson
Department of Soil and Crop Sciences, Texas A&M University, College Station, TX 77843, USA
Subas Malla
Department of Horticultural Sciences, Texas A&M University, College Station, TX 77843, USA; Texas A&M AgriLife Research and Extension Center, 1619 Garner Field Road, Uvalde, TX, 78801, USA; Corresponding author.
Anthracnose of watermelon is caused by a fungal pathogen Colletotrichum orbiculare. We generated F2 individuals from three different populations: Population 1 (PI 189225 x ‘New Hampshire Midget’), Population 2 (‘Perola’ x PI 189225), and Population 3 (‘Verona’ x PI 189225). The biparental F2 populations, parents and F1 individuals were inoculated with an isolate of race 2 anthracnose isolated from watermelon. Leaf lesions were visually rated seven days post inoculation on a scale of 0% (no lesion) to 100% (dead true leaf). Here we present the datasets obtained after the disease inoculation. The distribution of data obtained was visualized using histograms and goodness-of-fit was tested using Chi-Square. These datasets provide information on the mode of inheritance of race 2 anthracnose resistance in watermelon.
