Perturbation and resilience of the gut microbiome up to 3 months after β-lactams exposure in healthy volunteers suggest an important role of microbial β-lactamases
Camille d’Humières,
Margot Delavy,
Laurie Alla,
Farid Ichou,
Emilie Gauliard,
Amine Ghozlane,
Florence Levenez,
Nathalie Galleron,
Benoit Quinquis,
Nicolas Pons,
Jimmy Mullaert,
Antoine Bridier-Nahmias,
Bénédicte Condamine,
Marie Touchon,
Dominique Rainteau,
Antonin Lamazière,
Philippe Lesnik,
Maharajah Ponnaiah,
Marie Lhomme,
Natacha Sertour,
Savannah Devente,
Jean-Denis Docquier,
Marie-Elisabeth Bougnoux,
Olivier Tenaillon,
Mélanie Magnan,
Etienne Ruppé,
Nathalie Grall,
Xavier Duval,
Dusko Ehrlich,
France Mentré,
Erick Denamur,
Eduardo P. C. Rocha,
Emmanuelle Le Chatelier,
Charles Burdet,
for the PrediRes study group
Affiliations
Camille d’Humières
Université Paris Cité, IAME, INSERM
Margot Delavy
Institut Pasteur, Université Paris Cité, INRAE, USC2019, Unité Biologie Et Pathogénicité Fongiques
Laurie Alla
Université Paris-Saclay, INRAE, MetaGenoPolis
Farid Ichou
ICANomics, Foundation of Innovation in Cardiometabolism and Nutrition (IHU ICAN)
Emilie Gauliard
Sorbonne Université, INSERM U938, Centre de Recherche Saint-Antoine
Amine Ghozlane
Institut Pasteur, Université Paris Cité, Bioinformatics and Biostatistics Hub
Florence Levenez
Université Paris-Saclay, INRAE, MetaGenoPolis
Nathalie Galleron
Université Paris-Saclay, INRAE, MetaGenoPolis
Benoit Quinquis
Université Paris-Saclay, INRAE, MetaGenoPolis
Nicolas Pons
Université Paris-Saclay, INRAE, MetaGenoPolis
Jimmy Mullaert
Université Paris Cité, IAME, INSERM
Antoine Bridier-Nahmias
Université Paris Cité, IAME, INSERM
Bénédicte Condamine
Université Paris Cité, IAME, INSERM
Marie Touchon
Institut Pasteur, Université Paris Cité, CNRS UMR3525, Microbial Evolutionary Genomics
Dominique Rainteau
Sorbonne Université, INSERM U938, Centre de Recherche Saint-Antoine
Antonin Lamazière
Sorbonne Université, INSERM U938, Centre de Recherche Saint-Antoine
Philippe Lesnik
INSERM UMR-S 1166, Institute of Cardiometabolism and Nutrition, Sorbonne Université, Hôpital Pitié-Salpêtrière
Maharajah Ponnaiah
ICANomics, Foundation of Innovation in Cardiometabolism and Nutrition (IHU ICAN)
Marie Lhomme
ICANomics, Foundation of Innovation in Cardiometabolism and Nutrition (IHU ICAN)
Natacha Sertour
Institut Pasteur, Université Paris Cité, INRAE, USC2019, Unité Biologie Et Pathogénicité Fongiques
Savannah Devente
Dipartimento di Biotecnologie Mediche, Università di Siena
Jean-Denis Docquier
Dipartimento di Biotecnologie Mediche, Università di Siena
Marie-Elisabeth Bougnoux
Institut Pasteur, Université Paris Cité, INRAE, USC2019, Unité Biologie Et Pathogénicité Fongiques
Olivier Tenaillon
Université Paris Cité, IAME, INSERM
Mélanie Magnan
Université Paris Cité, IAME, INSERM
Etienne Ruppé
Université Paris Cité, IAME, INSERM
Nathalie Grall
Université Paris Cité, IAME, INSERM
Xavier Duval
Université Paris Cité, IAME, INSERM
Dusko Ehrlich
Université Paris-Saclay, INRAE, MetaGenoPolis
France Mentré
Université Paris Cité, IAME, INSERM
Erick Denamur
Université Paris Cité, IAME, INSERM
Eduardo P. C. Rocha
Institut Pasteur, Université Paris Cité, CNRS UMR3525, Microbial Evolutionary Genomics
Abstract Background Antibiotics notoriously perturb the gut microbiota. We treated healthy volunteers either with cefotaxime or ceftriaxone for 3 days, and collected in each subject 12 faecal samples up to day 90. Using untargeted and targeted phenotypic and genotypic approaches, we studied the changes in the bacterial, phage and fungal components of the microbiota as well as the metabolome and the β-lactamase activity of the stools. This allowed assessing their degrees of perturbation and resilience. Results While only two subjects had detectable concentrations of antibiotics in their faeces, suggesting important antibiotic degradation in the gut, the intravenous treatment perturbed very significantly the bacterial and phage microbiota, as well as the composition of the metabolome. In contrast, treatment impact was relatively low on the fungal microbiota. At the end of the surveillance period, we found evidence of resilience across the gut system since most components returned to a state like the initial one, even if the structure of the bacterial microbiota changed and the dynamics of the different components over time were rarely correlated. The observed richness of the antibiotic resistance genes repertoire was significantly reduced up to day 30, while a significant increase in the relative abundance of β-lactamase encoding genes was observed up to day 10, consistent with a concomitant increase in the β-lactamase activity of the microbiota. The level of β-lactamase activity at baseline was positively associated with the resilience of the metabolome content of the stools. Conclusions In healthy adults, antibiotics perturb many components of the microbiota, which return close to the baseline state within 30 days. These data suggest an important role of endogenous β-lactamase-producing anaerobes in protecting the functions of the microbiota by de-activating the antibiotics reaching the colon. Video Abstract