BMC Urology (Apr 2022)

A case report of acute testicular pain secondary to segmental testicular infarction

  • Hong-Liang Jin,
  • Qi Ma,
  • Jin Zhu,
  • Ya-Chen Zang,
  • Yi-Bin Zhou,
  • Bo-Xin Xue,
  • Dong-Rong Yang,
  • Chuan-Yang Sun,
  • Jie Gao,
  • Li-Jun Xu,
  • Bo Zhang

DOI
https://doi.org/10.1186/s12894-022-01006-7
Journal volume & issue
Vol. 22, no. 1
pp. 1 – 5

Abstract

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Abstract Background Segmental testicular infarction is a rare condition that often occurs in the upper pole of the left testicle and usually presents with acute onset of scrotal pain. Contrast-enhanced ultrasound and MR are essential for diagnosing and differentiating segmental testicular infarction in clinical practice, and conservative treatment can only be adopted after a definitive diagnosis. In the present case, after conservative treatment, the infarct volume was reduced, the blood flow around the infarct was increased, and blood flow signals appeared in the infarct. We performed a correlation analysis to investigate the causes of these changes. Case presentation A 33-year-old male, without any specific disease history, was admitted to the hospital with a 5-day history of left testicular pain, and the imaging showed focal necrosis of the left testicle with hemorrhage. He was diagnosed with segmental testicular infarction after differentiating and excluding it from malignant tumors. Conservative medical treatment was given, and the symptoms of testicular pain were relieved after treatment. After discharge, regular reexamination at follow-ups showed that the infarct’s size was reduced, the blood flow around the infarct was increased, and blood flow signals appeared in the infarct. Conclusion Conservative treatment has become the standard treatment currently adopted after confirming the diagnosis of segmental testicular infarction through contrast-enhanced ultrasound and MR. The blood flow changes in and around the focus of testicular infarction can be related to various factors. At present, relevant conclusions of the underlying mechanisms were mainly deduced from infarction studies of other related organs such as the heart and brain; thus, the specific pathological mechanism needs further experimental verification.

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