Frontiers in Immunology (Jun 2023)
The Wiskott–Aldrich syndrome protein is required for positive selection during T-cell lineage differentiation
- Melissa Pille,
- John Avila,
- Guillem Sanchez Sanchez,
- Guillem Sanchez Sanchez,
- Guillem Sanchez Sanchez,
- Guillem Sanchez Sanchez,
- Glenn Goetgeluk,
- Glenn Goetgeluk,
- Stijn De Munter,
- Stijn De Munter,
- Hanne Jansen,
- Lore Billiet,
- Karin Weening,
- Haipeng Xue,
- Sarah Bonte,
- Sarah Bonte,
- Joline Ingels,
- Joline Ingels,
- Laurenz De Cock,
- Laurenz De Cock,
- Eva Pascal,
- Eva Pascal,
- Lucas Deseins,
- Lucas Deseins,
- Tessa Kerre,
- Tessa Kerre,
- Tom Taghon,
- Tom Taghon,
- Georges Leclercq,
- Georges Leclercq,
- David Vermijlen,
- David Vermijlen,
- David Vermijlen,
- David Vermijlen,
- Brian Davis,
- Bart Vandekerckhove,
- Bart Vandekerckhove
Affiliations
- Melissa Pille
- Laboratory of Experimental Immunology, Department of Diagnostic Sciences, Ghent University, Ghent, Belgium
- John Avila
- Brown Foundation Institute of Molecular Medicine, Mc Govern Medical School, University of Texas Health Science Center at Houston, Houston, TX, United States
- Guillem Sanchez Sanchez
- Department of Pharmacotherapy and Pharmaceutics, Université Libre de Bruxelles (ULB), Brussels, Belgium
- Guillem Sanchez Sanchez
- Institute for Medical Immunology, Université Libre de Bruxelles (ULB), Brussels, Belgium
- Guillem Sanchez Sanchez
- ULB Center for Research in Immunology (U-CRI), Université Libre de Bruxelles (ULB), Brussels, Belgium
- Guillem Sanchez Sanchez
- WELBIO Department, WEL Research Institute, Wavre, Belgium
- Glenn Goetgeluk
- Laboratory of Experimental Immunology, Department of Diagnostic Sciences, Ghent University, Ghent, Belgium
- Glenn Goetgeluk
- Cancer Research Institute Ghent (CRIG), Ghent, Belgium
- Stijn De Munter
- Laboratory of Experimental Immunology, Department of Diagnostic Sciences, Ghent University, Ghent, Belgium
- Stijn De Munter
- Cancer Research Institute Ghent (CRIG), Ghent, Belgium
- Hanne Jansen
- Laboratory of Experimental Immunology, Department of Diagnostic Sciences, Ghent University, Ghent, Belgium
- Lore Billiet
- Laboratory of Experimental Immunology, Department of Diagnostic Sciences, Ghent University, Ghent, Belgium
- Karin Weening
- Laboratory of Experimental Immunology, Department of Diagnostic Sciences, Ghent University, Ghent, Belgium
- Haipeng Xue
- Brown Foundation Institute of Molecular Medicine, Mc Govern Medical School, University of Texas Health Science Center at Houston, Houston, TX, United States
- Sarah Bonte
- Cancer Research Institute Ghent (CRIG), Ghent, Belgium
- Sarah Bonte
- Department of Applied Mathematics, Computer Science and Statistics, Ghent University, Ghent, Belgium
- Joline Ingels
- Laboratory of Experimental Immunology, Department of Diagnostic Sciences, Ghent University, Ghent, Belgium
- Joline Ingels
- Cancer Research Institute Ghent (CRIG), Ghent, Belgium
- Laurenz De Cock
- Laboratory of Experimental Immunology, Department of Diagnostic Sciences, Ghent University, Ghent, Belgium
- Laurenz De Cock
- Cancer Research Institute Ghent (CRIG), Ghent, Belgium
- Eva Pascal
- Laboratory of Experimental Immunology, Department of Diagnostic Sciences, Ghent University, Ghent, Belgium
- Eva Pascal
- Cancer Research Institute Ghent (CRIG), Ghent, Belgium
- Lucas Deseins
- Laboratory of Experimental Immunology, Department of Diagnostic Sciences, Ghent University, Ghent, Belgium
- Lucas Deseins
- Cancer Research Institute Ghent (CRIG), Ghent, Belgium
- Tessa Kerre
- Department of Internal Medicine and Pediatrics, Ghent University Hospital, Ghent, Belgium
- Tessa Kerre
- 0Department of Hematology, Ghent University Hospital, Ghent, Belgium
- Tom Taghon
- Laboratory of Experimental Immunology, Department of Diagnostic Sciences, Ghent University, Ghent, Belgium
- Tom Taghon
- Cancer Research Institute Ghent (CRIG), Ghent, Belgium
- Georges Leclercq
- Laboratory of Experimental Immunology, Department of Diagnostic Sciences, Ghent University, Ghent, Belgium
- Georges Leclercq
- Cancer Research Institute Ghent (CRIG), Ghent, Belgium
- David Vermijlen
- Department of Pharmacotherapy and Pharmaceutics, Université Libre de Bruxelles (ULB), Brussels, Belgium
- David Vermijlen
- Institute for Medical Immunology, Université Libre de Bruxelles (ULB), Brussels, Belgium
- David Vermijlen
- ULB Center for Research in Immunology (U-CRI), Université Libre de Bruxelles (ULB), Brussels, Belgium
- David Vermijlen
- WELBIO Department, WEL Research Institute, Wavre, Belgium
- Brian Davis
- Brown Foundation Institute of Molecular Medicine, Mc Govern Medical School, University of Texas Health Science Center at Houston, Houston, TX, United States
- Bart Vandekerckhove
- Laboratory of Experimental Immunology, Department of Diagnostic Sciences, Ghent University, Ghent, Belgium
- Bart Vandekerckhove
- Cancer Research Institute Ghent (CRIG), Ghent, Belgium
- DOI
- https://doi.org/10.3389/fimmu.2023.1188099
- Journal volume & issue
-
Vol. 14
Abstract
The Wiskott–Aldrich syndrome (WAS) is an X-linked primary immune deficiency caused by a mutation in the WAS gene. This leads to altered or absent WAS protein (WASp) expression and function resulting in thrombocytopenia, eczema, recurrent infections, and autoimmunity. In T cells, WASp is required for immune synapse formation. Patients with WAS show reduced numbers of peripheral blood T lymphocytes and an altered T-cell receptor repertoire. In vitro, their peripheral T cells show decreased proliferation and cytokine production upon aCD3/aCD28 stimulation. It is unclear whether these T-cell defects are acquired during peripheral activation or are, in part, generated during thymic development. Here, we assessed the role of WASp during T-cell differentiation using artificial thymic organoid cultures and in the thymus of humanized mice. Although CRISPR/Cas9 WAS knockout hematopoietic stem and progenitor cells (HSPCs) rearranged the T-cell receptor and differentiated to T-cell receptor (TCR)+ CD4+ CD8+ double-positive (DP) cells similar to wild-type HSPCs, a partial defect in the generation of CD8 single-positive (SP) cells was observed, suggesting that WASp is involved in their positive selection. TCR repertoire analysis of the DP and CD8+ SP population, however, showed a polyclonal repertoire with no bias toward autoreactivity. To our knowledge, this is the first study of the role of WASp in human T-cell differentiation and on TCR repertoire generation.
Keywords