Long noncoding RNA MIAT inhibits the progression of diabetic nephropathy and the activation of NF-κB pathway in high glucose-treated renal tubular epithelial cells by the miR-182-5p/GPRC5A axis
Dong Qianlan,
Wang Qiong,
Yan Xiaohui,
Wang Xiaoming,
Li Zhenjiang,
Zhang Linping
Affiliations
Dong Qianlan
Kidney Disease and Dialysis Center, Shaanxi Provincial People’s Hospital, Beilin District, Xi’an, Shaanxi, 710068, China
Wang Qiong
Kidney Disease and Dialysis Center, Shaanxi Provincial People’s Hospital, Beilin District, Xi’an, Shaanxi, 710068, China
Yan Xiaohui
Kidney Disease and Dialysis Center, Shaanxi Provincial People’s Hospital, Beilin District, Xi’an, Shaanxi, 710068, China
Wang Xiaoming
Kidney Disease and Dialysis Center, Shaanxi Provincial People’s Hospital, Beilin District, Xi’an, Shaanxi, 710068, China
Li Zhenjiang
Kidney Disease and Dialysis Center, Shaanxi Provincial People’s Hospital, Beilin District, Xi’an, Shaanxi, 710068, China
Zhang Linping
Kidney Disease and Dialysis Center, Shaanxi Provincial People’s Hospital, Beilin District, Xi’an, Shaanxi, 710068, China
Diabetic nephropathy (DN) is a common diabetic complication. Long noncoding RNAs (lncRNAs) have been identified as essential regulators in DN progression. This study is devoted to the research of lncRNA-myocardial infarction-associated transcript (MIAT) in DN.
