Silicosis complicated with autoimmune pulmonary alveolar proteinosis caused by long-term dust inhalation during construction of bridge pier columns: A case report
Jie Jiang,
YouMing Zhu,
Tao Jiang,
TingTing Hu,
YiLing Gan
Affiliations
Jie Jiang
Department of Respiratory and Critical Care Medicine, Chongqing Prevention and Treatment Center for Occupational Diseases, No.301, Nancheng Avenue, Nanan District, Chongqing, 400060, PR China
YouMing Zhu
Department of Respiratory and Critical Care Medicine, Chongqing Prevention and Treatment Center for Occupational Diseases, No.301, Nancheng Avenue, Nanan District, Chongqing, 400060, PR China
Tao Jiang
Department of Respiratory and Critical Care Medicine, Chongqing Prevention and Treatment Center for Occupational Diseases, No.301, Nancheng Avenue, Nanan District, Chongqing, 400060, PR China
TingTing Hu
Department of Respiratory and Critical Care Medicine, Chongqing Prevention and Treatment Center for Occupational Diseases, No.301, Nancheng Avenue, Nanan District, Chongqing, 400060, PR China
YiLing Gan
Corresponding author. No.301, Nancheng Avenue, Nanan District, Chongqing, 400060, PR China.; Department of Respiratory and Critical Care Medicine, Chongqing Prevention and Treatment Center for Occupational Diseases, No.301, Nancheng Avenue, Nanan District, Chongqing, 400060, PR China
Pulmonary alveolar proteinosis (PAP) is characterized by the accumulation of surfactant material in alveoli. Few aPAP cases with a history of dust inhalation show both paves stone-like changes and micronodules in the chest CT scan. We present a 52-year-old male patient withsilicosis complicated with aPAP due to long-term dust inhalation during the construction of bridge piers columns. In this case report, chest CT of the patient displayed nonuniform ground-glass and patchy shadows in both lungs, paving stone-like changes, as well as diffuse distribution of high-density small nodular shadows, and the nodules tended to confluence.
