A Computational Study of the Shear Behavior of Reinforced Concrete Beams Affected from Alkali–Silica Reactivity Damage
Bora Gencturk,
Hadi Aryan,
Mohammad Hanifehzadeh,
Clotilde Chambreuil,
Jianqiang Wei
Affiliations
Bora Gencturk
Sonny Astani Department of Civil and Environmental Engineering, University of Southern California, 3620 S. Vermont Avenue, KAP 210, Los Angeles, CA 90089-2531, USA
Hadi Aryan
Sonny Astani Department of Civil and Environmental Engineering, University of Southern California, 3620 S. Vermont Avenue, KAP 210, Los Angeles, CA 90089-2531, USA
Mohammad Hanifehzadeh
Sonny Astani Department of Civil and Environmental Engineering, University of Southern California, 3620 S. Vermont Avenue, KAP 210, Los Angeles, CA 90089-2531, USA
Clotilde Chambreuil
LMT—Laboratoire de Mécanique et Technologie, University Paris-Saclay, ENS Paris-Saclay, CNRS, 91190 Gif-sur-Yvette, France
Jianqiang Wei
Department of Civil and Environmental Engineering, University of Massachusetts, Lowell, MA 01854-5104, USA
In this study, an investigation of the shear behavior of full-scale reinforced concrete (RC) beams affected from alkali–silica reactivity damage is presented. A detailed finite element model (FEM) was developed and validated with data obtained from the experiments using several metrics, including a force–deformation curve, rebar strains, and crack maps and width. The validated FEM was used in a parametric study to investigate the potential impact of alkali–silica reactivity (ASR) degradation on the shear capacity of the beam. Degradations of concrete mechanical properties were correlated with ASR expansion using material test data and implemented in the FEM for different expansions. The finite element (FE) analysis provided a better understanding of the failure mechanism of ASR-affected RC beam and degradation in the capacity as a function of the ASR expansion. The parametric study using the FEM showed 6%, 19%, and 25% reduction in the shear capacity of the beam, respectively, affected from 0.2%, 0.4%, and 0.6% of ASR-induced expansion.