Data in Brief
(Feb 2018)
Characterization of the transient middle cerebral artery occlusion model of ischemic stroke in a HuR transgenic mouse line
A. Ardelt,
R. Carpenter,
I. Iwuchukwu,
A. Zhang,
W. Lin,
E. Kosciuczuk,
C. Hinkson,
T. Rebeiz,
S. Reitz,
P. King
Affiliations
A. Ardelt
Department of Neurology, University of Chicago, 5841 S. Maryland Ave, MC2030, Chicago, IL 60637, USA; Corresponding author.
R. Carpenter
Department of Neurology, University of Chicago, 5841 S. Maryland Ave, MC2030, Chicago, IL 60637, USA
I. Iwuchukwu
Department of Neurocritical Care, Ochsner Medical Center, 1514 Jefferson Hwy., New Orleans, LA 70121, USA
A. Zhang
Department of Neurology, University of Chicago, 5841 S. Maryland Ave, MC2030, Chicago, IL 60637, USA
W. Lin
Department of Neurology, University of Chicago, 5841 S. Maryland Ave, MC2030, Chicago, IL 60637, USA
E. Kosciuczuk
Division of Hematology-Oncology, Northwestern University, 675 North St. Clair, Chicago, IL 60611, USA
C. Hinkson
Department of Neurology, University of Chicago, 5841 S. Maryland Ave, MC2030, Chicago, IL 60637, USA
T. Rebeiz
Department of Neurology, University of Chicago, 5841 S. Maryland Ave, MC2030, Chicago, IL 60637, USA
S. Reitz
Department of Neurology, University of Chicago, 5841 S. Maryland Ave, MC2030, Chicago, IL 60637, USA
P. King
Department of Neurology, University of Alabama at Birmingham, 1720 7th Sve South and the Birmingham VA Medical Center, Birmingham, AL 35233, USA
Journal volume & issue
Vol. 16
Abstract
Read online
This set of experiments characterizes a model of transient cerebral ischemic stroke in a transgenic (Tg) mouse line in which the glial fibrillary acidic protein (GFAP) promoter is utilized to drive expression of a human RNA-binding protein, HuR. Additionally, the effect of cerebral ischemia on the expression of endogenous Hu proteins is presented.
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