Disease Models & Mechanisms (Jun 2024)
Physiological stress improves stem cell modeling of dystrophic cardiomyopathy
- Dominic E. Fullenkamp,
- Alexander B. Willis,
- Jodi L. Curtin,
- Ansel P. Amaral,
- Kyle T. Dittloff,
- Sloane I. Harris,
- Ivana A. Chychula,
- Cory W. Holgren,
- Paul W. Burridge,
- Brenda Russell,
- Alexis R. Demonbreun,
- Elizabeth M. McNally
Affiliations
- Dominic E. Fullenkamp
- Center for Genetic Medicine, Feinberg School of Medicine, Northwestern University, Chicago, IL 60611, USA
- Alexander B. Willis
- Center for Genetic Medicine, Feinberg School of Medicine, Northwestern University, Chicago, IL 60611, USA
- Jodi L. Curtin
- Center for Genetic Medicine, Feinberg School of Medicine, Northwestern University, Chicago, IL 60611, USA
- Ansel P. Amaral
- Division of Cardiology, Department of Medicine, Feinberg School of Medicine, Northwestern University, Chicago, IL 60611, USA
- Kyle T. Dittloff
- Department of Physiology and Biophysics, University of Illinois at Chicago, Chicago, IL 60612, USA
- Sloane I. Harris
- Center for Genetic Medicine, Feinberg School of Medicine, Northwestern University, Chicago, IL 60611, USA
- Ivana A. Chychula
- Center for Genetic Medicine, Feinberg School of Medicine, Northwestern University, Chicago, IL 60611, USA
- Cory W. Holgren
- Center for Genetic Medicine, Feinberg School of Medicine, Northwestern University, Chicago, IL 60611, USA
- Paul W. Burridge
- Department of Pharmacology, Feinberg School of Medicine, Northwestern University, Chicago, IL 60611, USA
- Brenda Russell
- Department of Physiology and Biophysics, University of Illinois at Chicago, Chicago, IL 60612, USA
- Alexis R. Demonbreun
- Center for Genetic Medicine, Feinberg School of Medicine, Northwestern University, Chicago, IL 60611, USA
- Elizabeth M. McNally
- Center for Genetic Medicine, Feinberg School of Medicine, Northwestern University, Chicago, IL 60611, USA
- DOI
- https://doi.org/10.1242/dmm.050487
- Journal volume & issue
-
Vol. 17,
no. 6
Abstract
No abstracts available.Keywords
- membrane
- annexin
- sarcolemma
- cardiomyocyte
- human induced pluripotent stem cell-derived cardiomyocytes
- duchenne muscular dystrophy