TRPV3-ANO1 interaction positively regulates wound healing in keratinocytes

Yu Yamanoi; Jing Lei; Yasunori Takayama; Shigekuni Hosogi; Yoshinori Marunaka; Makoto Tominaga

doi:10.1038/s42003-023-04482-1

Communications Biology (Jan 2023)

TRPV3-ANO1 interaction positively regulates wound healing in keratinocytes

Yu Yamanoi,
Jing Lei,
Yasunori Takayama,
Shigekuni Hosogi,
Yoshinori Marunaka,
Makoto Tominaga

Affiliations

Yu Yamanoi: Thermal Biology Group, Exploratory Research Center on Life and Living Systems (ExCELLS)
Jing Lei: Thermal Biology Group, Exploratory Research Center on Life and Living Systems (ExCELLS)
Yasunori Takayama: Department of Physiology, Showa University School of Medicine
Shigekuni Hosogi: Department of Clinical and Translational Physiology, Kyoto Pharmaceutical University
Yoshinori Marunaka: Research Organization of Science and Technology, Ritsumeikan University
Makoto Tominaga: Thermal Biology Group, Exploratory Research Center on Life and Living Systems (ExCELLS)

DOI: https://doi.org/10.1038/s42003-023-04482-1
Journal volume & issue: Vol. 6, no. 1
pp. 1 – 13

Abstract

Read online

The chloride channel anoctamin1, upon activation by calcium influx through transient receptor potential vanilloid 3, mediates chloride influx and enhances wound healing in keratinocytes.

Published in Communications Biology

ISSN: 2399-3642 (Online)
Publisher: Nature Portfolio
Country of publisher: United Kingdom
LCC subjects: Science: Biology (General)
Website: https://www.nature.com/commsbio/

About the journal