Normal-Tension Glaucoma: Epidemiology, Pathogenesis, Diagnosis and Treatment. Review. Part 1
N. I. Kurysheva,
V. E. Kim,
H. M. Plieva,
V. Yu. Kim,
D. M. Nurmahadzhiev
Affiliations
N. I. Kurysheva
Medical Biological University of Innovations and Continuing Education of the Federal Biophysical Center named after A.I. Burnazyan; Diagnostic Department of the Ophthalmological Center of Federal Medical-Biological Agency; Department of Ophthalmology of the Postgraduate Education Academy of the Burnasyan Federal Medical Biophysical Center of the Federal Medical Biological Agency
V. E. Kim
Medical Biological University of Innovations and Continuing Education of the Federal Biophysical Center named after A.I. Burnazyan; Diagnostic Department of the Ophthalmological Center of Federal Medical-Biological Agency
H. M. Plieva
Medical Biological University of Innovations and Continuing Education of the Federal Biophysical Center named after A.I. Burnazyan; Diagnostic Department of the Ophthalmological Center of Federal Medical-Biological Agency
V. Yu. Kim
Medical Biological University of Innovations and Continuing Education of the Federal Biophysical Center named after A.I. Burnazyan; Diagnostic Department of the Ophthalmological Center of Federal Medical-Biological Agency
D. M. Nurmahadzhiev
Medical Biological University of Innovations and Continuing Education of the Federal Biophysical Center named after A.I. Burnazyan
In the modern world, glaucoma is one of the main causes of irreversible vision loss. Normal tension glaucoma (NTG) constitutes the major proportion of POAG, which is common in Asian populations. NTG is a disease resulted from the interaction of multiple genetic risk factors. But other factors are also important, such as thinning of the scleral lamina cribrosa and a lesion of the transmembrane pressure gradient. Central visual field defects and disc hemorrhage are characteristics finding that is highly associated with the progression of NTG.
