Molecular Mimicry between Meningococcal B Factor H-Binding Protein and Human Proteins

Abstract

This study calls attention on molecular mimicry and the consequent autoimmune cross reactivity as the molecular mechanism that can cause adverse events following meningococcal B vaccination and warns against active immunizations based on entire antigen.

Keywords

• molecular mimicry
• Trumenba vaccine
• factor H-binding protein
• human proteome
• adverse events