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Disease Models & Mechanisms
(Jul 2023)
A zebrafish model of combined saposin deficiency identifies acid sphingomyelinase as a potential therapeutic target
Tejia Zhang,
Ivy Alonzo,
Chris Stubben,
Yijie Geng,
Chelsea Herdman,
Nancy Chandler,
Kim P. Doane,
Brock R. Pluimer,
Sunia A. Trauger,
Randall T. Peterson
Affiliations
Tejia Zhang
Department of Pharmacology and Toxicology, University of Utah, Salt Lake City, UT 84112, USA
Ivy Alonzo
Department of Pharmacology and Toxicology, University of Utah, Salt Lake City, UT 84112, USA
Chris Stubben
Bioinformatic Analysis Shared Resource, Huntsman Cancer Institute, University of Utah, Salt Lake City, UT 84112, USA
Yijie Geng
Department of Pharmacology and Toxicology, University of Utah, Salt Lake City, UT 84112, USA
Chelsea Herdman
Department of Neurobiology and Molecular Medicine Program, University of Utah School of Medicine, Salt Lake City, UT 84112, USA
Nancy Chandler
Electron Microscopy Core Laboratory, University of Utah, Salt Lake City, UT 84112, USA
Kim P. Doane
Department of Pharmacology and Toxicology, University of Utah, Salt Lake City, UT 84112, USA
Brock R. Pluimer
Department of Pharmacology and Toxicology, University of Utah, Salt Lake City, UT 84112, USA
Sunia A. Trauger
Harvard Center for Mass Spectrometry, Harvard University, Cambridge, MA 02138, USA
Randall T. Peterson
Department of Pharmacology and Toxicology, University of Utah, Salt Lake City, UT 84112, USA
DOI
https://doi.org/10.1242/dmm.049995
Journal volume & issue
Vol. 16, no. 7
Abstract
Read online
No abstracts available.
Keywords
lipidomics
lysosomal storage disease
zebrafish
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