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Disease Models & Mechanisms
(Sep 2024)
The highly metastatic 4T1 breast carcinoma model possesses features of a hybrid epithelial/mesenchymal phenotype
Mary E. Herndon,
Mitchell Ayers,
Katherine N. Gibson-Corley,
Michael K. Wendt,
Lori L. Wallrath,
Michael D. Henry,
Christopher S. Stipp
Affiliations
Mary E. Herndon
Department of Biology, University of Iowa, Iowa City, IA 52245, USA
Mitchell Ayers
Department of Medicinal Chemistry and Molecular Pharmacology, Purdue University, West Lafayette, IN 47907, USA
Katherine N. Gibson-Corley
Department of Pathology, Microbiology and Immunology, Vanderbilt University Medical Center, Nashville, TN 37232, USA
Michael K. Wendt
Holden Comprehensive Cancer Center, University of Iowa, Iowa City, IA 52242, USA
Lori L. Wallrath
Holden Comprehensive Cancer Center, University of Iowa, Iowa City, IA 52242, USA
Michael D. Henry
Holden Comprehensive Cancer Center, University of Iowa, Iowa City, IA 52242, USA
Christopher S. Stipp
Department of Biology, University of Iowa, Iowa City, IA 52245, USA
DOI
https://doi.org/10.1242/dmm.050771
Journal volume & issue
Vol. 17, no. 9
Abstract
Read online
No abstracts available.
Keywords
e-cadherin
breast cancer
collective migration
epithelial-mesenchymal transition
metastasis
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