Neural Regeneration Research (Jan 2015)
Shrinkage of ipsilateral taste buds and hyperplasia of contralateral taste buds following chorda tympani nerve transection
- Yi-ke Li,
- Juan-mei Yang,
- Yi-bo Huang,
- Dong-dong Ren,
- Fang-lu Chi
Affiliations
- Yi-ke Li
- Juan-mei Yang
- Yi-bo Huang
- Dong-dong Ren
- Fang-lu Chi
- DOI
- https://doi.org/10.4103/1673-5374.158366
- Journal volume & issue
-
Vol. 10,
no. 6
pp. 989 – 995
Abstract
The morphological changes that occur in the taste buds after denervation are not well understood in rats, especially in the contralateral tongue epithelium. In this study, we investigated the time course of morphological changes in the taste buds following unilateral nerve transection. The role of the trigeminal component of the lingual nerve in maintaining the structural integrity of the taste buds was also examined. Twenty-four Sprague-Dawley rats were randomly divided into three groups: control, unilateral chorda tympani nerve transection and unilateral chorda tympani nerve transection + lingual nerve transection. Rats were allowed up to 42 days of recovery before being euthanized. The taste buds were visualized using a cytokeratin 8 antibody. Taste bud counts, volumes and taste receptor cell numbers were quantified and compared among groups. No significant difference was detected between the chorda tympani nerve transection and chorda tympani nerve transection + lingual nerve transection groups. Taste bud counts, volumes and taste receptor cell numbers on the ipsilateral side all decreased significantly compared with control. On the contralateral side, the number of taste buds remained unchanged over time, but they were larger, and taste receptor cells were more numerous postoperatively. There was no evidence for a role of the trigeminal branch of the lingual nerve in maintaining the structural integrity of the anterior taste buds.
Keywords
- microtubule
- axon
- kinesin-5
- Eg5
- regeneration
- monastrol
- molecular motor protein
- aging
- neurodegenerative disorders
- telomere shortening
- MSCs
- cellular therapy
- traumatic brain injury
- spinal cord injuries
- dual diagnosis
- diagnosis
- complications
- rehabilitation
- post-concussion syndrome
- brain concussion
- blood brain barrier
- phage display
- peptide library
- nanocarrier
- targeting
- Schwann cells
- neurite outgrowth
- neuromuscular junction (NMJ)
- multiple sclerosis
- TGF-β/BMP-7/Smad signaling
- myogenic differentiation
- Trf3
- tumor suppression
- nerve regeneration
- bone marrow mesenchymal stem cells
- cerebral ischemia
- tail vein injection
- middle cerebral artery occlusion
- cell therapy
- neuroprotection
- nerve regeneration
- brain injury
- neuroimaging
- ferumoxytol
- superparamagnetic iron oxide particles
- human adipose-derived stem cells
- middle cerebral artery occlusion
- intracerebral injection
- magnetic resonance imaging
- enhanced susceptibility-weighted angiography image
- modified neurological severity scores
- rats
- Prussian blue staining
- neural regeneration
- neural regeneration
- non-invasive brain stimulation
- transcranial magnetic stimulation
- neurotrophic factor
- brain-derived neurotrophic factor
- neuroplasticity
- hippocampus
- aging
- cognitive function
- nerve regeneration
- curcumin
- neurons
- HIV-1 gp120 V3 loop
- plasticity
- HIV-associated neurocognitive disorders
- output/input curve
- long-term potentiation
- excitatory postsynaptic potential
- paired-pulse facilitation
- Ca 2+
- synaptosome
- NSFC grants
- neural regeneration
- nerve regeneration
- brain injury
- hydrogen sulfide
- cerebral ischemia/reperfusion injury
- P2X 7 receptor
- 2
- 3
- 5-triphenyl-2H-tetrazolium chloride staining
- animal model
- protection
- sodium hydrosulfide
- immunofluorescence
- middle cerebral artery occlusion
- NSFC grant
- neural regeneration
- nerve regeneration
- γ-aminobutyric acid
- glial fibrillary acidic protein
- glutamic acid decarboxylase
- neurotoxicity
- weaning
- organ index
- cerebrum
- cortex
- glutamate
- neural regeneration
- p53 tumor suppressor gene family
- cerebral ischemia/reperfusion
- pyramidal neurons
- CA1 region
- delayed neuronal death
- immunohistochemistry
- western blotting
- neural regeneration
- nerve regeneration
- spinal cord injury
- rapamycin
- Wnt/β-catenin signaling pathway
- apoptosis
- caspase-3
- brain-derived neurotrophic factor
- neuroprotection
- loss of neurons
- NSFC grants
- neural regeneration
- nerve regeneration
- spinal cord injury
- hydrogen-rich saline
- reactive oxygen species
- physiological saline
- oxidative stress
- Basso
- Beattie and Bresnahan score
- malondialdehyde
- superoxide dismutase
- calcitonin gene-related peptide
- caspase-3
- neural regeneration
- nerve regeneration
- peripheral nerve injury
- rabbits
- sciatic nerve injury
- autologous nerve repair
- polylactic glycolic acid conduit
- extracellular matrix gel
- grafting
- stress relaxation
- creep
- viscoelasticity
- histomorphology
- electrophysiology
- neural regeneration
- nerve regeneration
- peripheral nerve injury
- pain sense model
- dorsal root ganglion
- primary sensory neuron
- glycosylated membrane protein
- sialic acid
- cell electrophoresis
- electrophoresis velocity
- heat-hyperalgesia behavior
- hyperalgesia
- neuraminidase
- neural regeneration
- N-propionylmannosamine
- sialic acid
- glycoengineering
- sciatic nerve
- peripheral nerve
- branching
- arborisation
- Thy1-YFP mouse
- nerve regeneration
- stem cells
- optic nerve
- optic neuropathy
- ophthalmology
- bone marrow-derived stem cells
- blindness
- visual loss
- nerve regeneration
- gustation
- cytokeratin
- tongue epithelium
- immunohistochemistry
- taste bud
- trigeminal nerve disorder
- NSFC grants
- neural regeneration