Impaired bone healing in type 2 diabetes is caused by defective bone microenvironment functions of skeletal progenitor cells
Florence Figeac,
Michaela Tencerova,
Dalia Ali,
Thomas L. Andersen,
Dan Rémi Christiansen Appadoo,
Nicholas Ditzel,
Justyna Magdalena Kowal,
Moustapha Kassem
Affiliations
Florence Figeac
Molecular Endocrinology, KMEB University of Southern Denmark and Odense University Hospital, Odense, Denmark
Michaela Tencerova
Molecular Endocrinology, KMEB University of Southern Denmark and Odense University Hospital, Odense, Denmark; Molecular Physiology of Bone, Institute of Physiology, Czech Academy of Sciences, Prague, Czech Republic
Dalia Ali
Molecular Endocrinology, KMEB University of Southern Denmark and Odense University Hospital, Odense, Denmark
Thomas L. Andersen
Pathology, Clinical Cell Biology- Odense University Hospital, Odense, Denmark; Clinical Research, University of Southern Denmark, Odense, Denmark; Molecular Medicine, University of Southern Denmark, Odense, Denmark
Dan Rémi Christiansen Appadoo
Molecular Endocrinology, KMEB University of Southern Denmark and Odense University Hospital, Odense, Denmark
Nicholas Ditzel
Molecular Endocrinology, KMEB University of Southern Denmark and Odense University Hospital, Odense, Denmark
Justyna Magdalena Kowal
Molecular Endocrinology, KMEB University of Southern Denmark and Odense University Hospital, Odense, Denmark
Moustapha Kassem
Molecular Endocrinology, KMEB University of Southern Denmark and Odense University Hospital, Odense, Denmark; Cellular and Molecular Medicine, Danish Stem Cell Center (DanStem)- University of Copenhagen, Copenhagen, Denmark; Corresponding author at: Molecular Endocrinology, KMEB University of Southern Denmark and Odense University Hospital, Odense, Denmark.
