Data in Brief (Jun 2016)

Data on necrotic and apoptotic cell death in acute myocardial ischemia/reperfusion injury: the effects of CaMKII and angiotensin AT1 receptor inhibition

  • Tomas Rajtik,
  • Slavka Carnicka,
  • Adrian Szobi,
  • Zoltan Giricz,
  • Jin O-Uchi,
  • Veronika Hassova,
  • Pavel Svec,
  • Peter Ferdinandy,
  • Tanya Ravingerova,
  • Adriana Adameova

Journal volume & issue
Vol. 7
pp. 730 – 734

Abstract

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Content of particular proteins indicating cellular injury due to apoptosis and necrosis has been investigated in ischemic/reperfused (IR) hearts and ischemic/reperfused hearts treated with CaMKII inhibitor and/or AT1 receptor inhibitor. This data article provides information in support of the original research article “Oxidative activation of CaMKIIδ in acute myocardial ischemia/reperfusion injury: a role of angiotensin AT1 receptor-NOX2 signaling axis” [1]. Keywords: Myocardial ischemia/reperfusion injury, CaMKII inhibition, AT1 receptor blockade, Apoptosis, Necrosis