PLoS Genetics (Feb 2014)
Fine-mapping the HOXB region detects common variants tagging a rare coding allele: evidence for synthetic association in prostate cancer.
- Edward J Saunders,
- Tokhir Dadaev,
- Daniel A Leongamornlert,
- Sarah Jugurnauth-Little,
- Malgorzata Tymrakiewicz,
- Fredrik Wiklund,
- Ali Amin Al Olama,
- Sara Benlloch,
- David E Neal,
- Freddie C Hamdy,
- Jenny L Donovan,
- Graham G Giles,
- Gianluca Severi,
- Henrik Gronberg,
- Markus Aly,
- Christopher A Haiman,
- Fredrick Schumacher,
- Brian E Henderson,
- Sara Lindstrom,
- Peter Kraft,
- David J Hunter,
- Susan Gapstur,
- Stephen Chanock,
- Sonja I Berndt,
- Demetrius Albanes,
- Gerald Andriole,
- Johanna Schleutker,
- Maren Weischer,
- Børge G Nordestgaard,
- Federico Canzian,
- Daniele Campa,
- Elio Riboli,
- Tim J Key,
- Ruth C Travis,
- Sue A Ingles,
- Esther M John,
- Richard B Hayes,
- Paul Pharoah,
- Kay-Tee Khaw,
- Janet L Stanford,
- Elaine A Ostrander,
- Lisa B Signorello,
- Stephen N Thibodeau,
- Daniel Schaid,
- Christiane Maier,
- Adam S Kibel,
- Cezary Cybulski,
- Lisa Cannon-Albright,
- Hermann Brenner,
- Jong Y Park,
- Radka Kaneva,
- Jyotsna Batra,
- Judith A Clements,
- Manuel R Teixeira,
- Jianfeng Xu,
- Christos Mikropoulos,
- Chee Goh,
- Koveela Govindasami,
- Michelle Guy,
- Rosemary A Wilkinson,
- Emma J Sawyer,
- Angela Morgan,
- COGS-CRUK GWAS-ELLIPSE (Part of GAME-ON) Initiative,
- UK Genetic Prostate Cancer Study Collaborators,
- UK ProtecT Study Collaborators,
- PRACTICAL Consortium,
- Douglas F Easton,
- Ken Muir,
- Rosalind A Eeles,
- Zsofia Kote-Jarai
Affiliations
- Edward J Saunders
- Tokhir Dadaev
- Daniel A Leongamornlert
- Sarah Jugurnauth-Little
- Malgorzata Tymrakiewicz
- Fredrik Wiklund
- Ali Amin Al Olama
- Sara Benlloch
- David E Neal
- Freddie C Hamdy
- Jenny L Donovan
- Graham G Giles
- Gianluca Severi
- Henrik Gronberg
- Markus Aly
- Christopher A Haiman
- Fredrick Schumacher
- Brian E Henderson
- Sara Lindstrom
- Peter Kraft
- David J Hunter
- Susan Gapstur
- Stephen Chanock
- Sonja I Berndt
- Demetrius Albanes
- Gerald Andriole
- Johanna Schleutker
- Maren Weischer
- Børge G Nordestgaard
- Federico Canzian
- Daniele Campa
- Elio Riboli
- Tim J Key
- Ruth C Travis
- Sue A Ingles
- Esther M John
- Richard B Hayes
- Paul Pharoah
- Kay-Tee Khaw
- Janet L Stanford
- Elaine A Ostrander
- Lisa B Signorello
- Stephen N Thibodeau
- Daniel Schaid
- Christiane Maier
- Adam S Kibel
- Cezary Cybulski
- Lisa Cannon-Albright
- Hermann Brenner
- Jong Y Park
- Radka Kaneva
- Jyotsna Batra
- Judith A Clements
- Manuel R Teixeira
- Jianfeng Xu
- Christos Mikropoulos
- Chee Goh
- Koveela Govindasami
- Michelle Guy
- Rosemary A Wilkinson
- Emma J Sawyer
- Angela Morgan
- COGS-CRUK GWAS-ELLIPSE (Part of GAME-ON) Initiative
- UK Genetic Prostate Cancer Study Collaborators
- UK ProtecT Study Collaborators
- PRACTICAL Consortium
- Douglas F Easton
- Ken Muir
- Rosalind A Eeles
- Zsofia Kote-Jarai
- DOI
- https://doi.org/10.1371/journal.pgen.1004129
- Journal volume & issue
-
Vol. 10,
no. 2
p. e1004129
Abstract
The HOXB13 gene has been implicated in prostate cancer (PrCa) susceptibility. We performed a high resolution fine-mapping analysis to comprehensively evaluate the association between common genetic variation across the HOXB genetic locus at 17q21 and PrCa risk. This involved genotyping 700 SNPs using a custom Illumina iSelect array (iCOGS) followed by imputation of 3195 SNPs in 20,440 PrCa cases and 21,469 controls in The PRACTICAL consortium. We identified a cluster of highly correlated common variants situated within or closely upstream of HOXB13 that were significantly associated with PrCa risk, described by rs117576373 (OR 1.30, P = 2.62×10(-14)). Additional genotyping, conditional regression and haplotype analyses indicated that the newly identified common variants tag a rare, partially correlated coding variant in the HOXB13 gene (G84E, rs138213197), which has been identified recently as a moderate penetrance PrCa susceptibility allele. The potential for GWAS associations detected through common SNPs to be driven by rare causal variants with higher relative risks has long been proposed; however, to our knowledge this is the first experimental evidence for this phenomenon of synthetic association contributing to cancer susceptibility.