Alzheimer’s & Dementia: Diagnosis, Assessment & Disease Monitoring (Oct 2023)
Cross‐sectional and longitudinal evaluation of plasma glial fibrillary acidic protein to detect and predict clinical syndromes of Alzheimer's disease
- Madeline Ally,
- Michael A. Sugarman,
- Henrik Zetterberg,
- Kaj Blennow,
- Nicholas J. Ashton,
- Thomas K. Karikari,
- Hugo J. Aparicio,
- Brandon Frank,
- Yorghos Tripodis,
- Brett Martin,
- Joseph N. Palmisano,
- Eric G. Steinberg,
- Irene Simkin,
- Lindsay A. Farrer,
- Gyungah R. Jun,
- Katherine W. Turk,
- Andrew E. Budson,
- Maureen K. O'Connor,
- Rhoda Au,
- Lee E. Goldstein,
- Neil W. Kowall,
- Ronald Killiany,
- Robert A. Stern,
- Thor D. Stein,
- Ann C. McKee,
- Wei Qiao Qiu,
- Jesse Mez,
- Michael L. Alosco
Affiliations
- Madeline Ally
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Michael A. Sugarman
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Henrik Zetterberg
- Department of Neurodegenerative Disease UCL Institute of Neurology London UK
- Kaj Blennow
- Clinical Neurochemistry Laboratory Sahlgrenska University Hospital Mölndal Sweden
- Nicholas J. Ashton
- Department of Psychiatry and Neurochemistry, Institute of Neuroscience and Physiology The Sahlgrenska Academy at the University of Gothenburg Gothenburg Sweden
- Thomas K. Karikari
- Clinical Neurochemistry Laboratory Sahlgrenska University Hospital Mölndal Sweden
- Hugo J. Aparicio
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Brandon Frank
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Yorghos Tripodis
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Brett Martin
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Joseph N. Palmisano
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Eric G. Steinberg
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Irene Simkin
- Department of Medicine Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Lindsay A. Farrer
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Gyungah R. Jun
- Department of Medicine Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Katherine W. Turk
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Andrew E. Budson
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Maureen K. O'Connor
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Rhoda Au
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Lee E. Goldstein
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Neil W. Kowall
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Ronald Killiany
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Robert A. Stern
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Thor D. Stein
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Ann C. McKee
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Wei Qiao Qiu
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Jesse Mez
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- Michael L. Alosco
- Boston University Alzheimer's Disease Research Center and CTE Center Boston University Chobanian & Avedisian School of Medicine Boston Massachusetts USA
- DOI
- https://doi.org/10.1002/dad2.12492
- Journal volume & issue
-
Vol. 15,
no. 4
pp. n/a – n/a
Abstract
Abstract Introduction This study examined plasma glial fibrillary acidic protein (GFAP) as a biomarker of cognitive impairment due to Alzheimer's disease (AD) with and against plasma neurofilament light chain (NfL), and phosphorylated tau (p‐tau)181+231. Methods Plasma samples were analyzed using Simoa platform for 567 participants spanning the AD continuum. Cognitive diagnosis, neuropsychological testing, and dementia severity were examined for cross‐sectional and longitudinal outcomes. Results Plasma GFAP discriminated AD dementia from normal cognition (adjusted mean difference = 0.90 standard deviation [SD]) and mild cognitive impairment (adjusted mean difference = 0.72 SD), and demonstrated superior discrimination compared to alternative plasma biomarkers. Higher GFAP was associated with worse dementia severity and worse performance on 11 of 12 neuropsychological tests. Longitudinally, GFAP predicted decline in memory, but did not predict conversion to mild cognitive impairment or dementia. Discussion Plasma GFAP was associated with clinical outcomes related to suspected AD and could be of assistance in a plasma biomarker panel to detect in vivo AD.
Keywords
- Alzheimer's disease
- glial fibrillary acidic protein
- neuropsychology
- neurofilament light chain
- phosphorylated tau
- plasma biomarkers